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In nerves exposed to a hundred Hz when bathed in 10 mM glucose, the CAP failed suggesting that even in the presence of normoglycemic glucose additional power substrate is required when the tissue energy demand increases. Such knowledge are vital as they present that glycogen has an important position beneath physiological circumstances. The truth that glycogen supported CAP conduction in the absence of exogenously applied glucose most likely has no worth in ascribing a task to glycogen, since aglycaemia is an artificial experimental procedure, and hypoglycemia is an iatrogenic consequence of insulin therapy that didn't exist previous to 1922 (Frier et al., 2014), thus cannot be glycogen’s foremost role. However supplying lactate to axons when tissue vitality demand increases implies a novel position for brain glycogen and suggests it acts as an power buffer fairly than an energy reserve, whose position is not to single-handedly support conduction, however to act in concert with ambient ranges of normoglycemic glucose.

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